From Mechanism to Medicine: What Drugs That Reached the Clinic Have in Common — and What Held Others Back

What 2,500+ studies reveal about the relationship between in vivo data quality and translational success – and what it means for the next generation of…

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PhenoMaster - Polyagonist Therapies in Obesity

Polyagonist Therapies in Obesity: A systems-level case study enabled by continuous in vivo phenotyping

Introduction  Obesity and type 2 diabetes are not single-pathway diseases. They emerge from a complex disruption of whole-body energy homeostasis involving the gut, brain, pancreas,…

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Learn how cage-level humidity and temperature shape thermogenesis, energy expenditure and metabolic outcomes in rodent studies. Real data, mechanisms and insights.

Micro-Environmental Humidity and Temperature in Rodent Metabolic Research

by Pierre-Louis Ruffault, PhD Cage-level humidity and temperature in rodent metabolic research play a critical yet often underestimated role in shaping thermogenesis, energy expenditure, and…

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Scientific Insights

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LinkedIn post by TSE Systems about behavioral phenotyping. Image shows gloved hands holding a pipette over a sample in a laboratory setting. Text reads: 'The Best Data Comes From Animals That Don't Know They're Being Tested' with subtitle 'A principle for behavioral phenotyping that changes what you measure.' Cyan 'Best Practices' badge in top right corner

How Handling Stress Contaminates Behavioral Data and What to do Instead

There is a confounder in almost every behavioral neuroscience study that nobody mentioned  in the methods section. Handling stress. The moment you pick up an…

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**Alt text:** Schematic summary of a 2026 Molecular Cell study from Prof. Christian Wolfrum’s lab describing MEDAG as a molecular regulator of adipocyte metabolism. The figure illustrates MEDAG (mesenteric estrogen-dependent adipogenesis gene) acting as an A-kinase anchoring protein (AKAP) that binds the regulatory subunit PKA-RIIβ to organize protein kinase A (PKA) signaling complexes in fat cells. A feedback loop is shown in which activated PKA phosphorylates MEDAG, and phosphorylated MEDAG helps restrain further PKA activity, limiting signaling intensity. In adipocyte-specific Medag knockout mice, loss of this restraint leads to increased PKA activity, higher energy expenditure, protection from diet-induced obesity, and a shift toward increased carbohydrate utilization without changes in food intake or physical activity. Whole-body metabolic changes were measured using indirect calorimetry in the PhenoMaster system from TSE Systems, confirming increased energy dissipation in adipose tissue.

A Newly Discovered Molecular Brake on Fat Metabolism: The Role of MEDAG

A recent study from Prof. Christian Wolfrum’s lab, published in Molecular Cell (2026), provides compelling new insights into how adipose tissue regulates energy balance. At…

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Maternal Immune Activation as a Neurodevelopmental Risk Factor: New Insights into ADHD-Like Endophenotypes in a Mouse Model

Maternal Immune Activation as a Neurodevelopmental Risk Factor: New Insights into ADHD-Like Endophenotypes in a Mouse Model

Overview Epidemiological evidence consistently links maternal infection during pregnancy to elevated offspring risk for neurodevelopmental disorders, including ADHD, schizophrenia, and autism spectrum disorder. Yet the…

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BonnBrain 2026

BonnBrain 2026

We are excited to share that we will participate in BonnBrain 2026 – From Genes to Circuits and Behavior, taking place March 23–25, 2026 at…

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American Physiology Summit 2025

Meet us at booth #1314 in American Physiology Summit from April 24-27 in Baltimore. Held annually by the American Physiological Society (APS), the Summit convenes…

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Neuroscience TSE Systems

Fens Forum 2026

Join TSE Systems at booth booth #98 and thousands of neuroscientists in Barcelona from 6–10 July 2026 for Europe’s premier international neuroscience meeting. By submitting your abstract,…

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